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The mostcommon variants associated with many different types of human cancers (and miss-ing the p53 interacting domain) are: mdm2-a (lacking exons 4–9), mdm2-b (lackingexons 4–11), and mdm2-c (lacking exons 5–9) (see Table 14.2).

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This is particularly applicable tocentrally acting drugs, e.g. We generated a rabbit polyclonalantibody to the MDM2-C isoform. This interaction of p53 with RAD51 and subsequent inhibition ofrecombination was further conrmed by overexpressing mutant L186PRAD51 thatprevented p53 binding to RAD51 [43]. Ofnote, pravastatin has been approved for childrengreater than 8 years of age.

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